Studies of the development of congenital anomalies in embryos of riboflavin‐deficient, galactoflavin fed rats. II. Role of the terminal electron transport systems
作者:
Oguz Aksu,
Bruce Mackler,
Thomas H. Shepard,
Ronald J. Lemire,
期刊:
Teratology
(WILEY Available online 1968)
卷期:
Volume 1,
issue 1
页码: 93-102
ISSN:0040-3709
年代: 1968
DOI:10.1002/tera.1420010110
出版商: Wiley Subscription Services, Inc., A Wiley Company
数据来源: WILEY
摘要:
AbstractStudies of the tissues of embryos and fetuses of rats treated with a riboflavin‐deficient, galactoflavin‐augmented diet during pregnancy were performed, and demonstrated that the activities of the succinic and DPNH oxidase systems (the terminal electron transport systems) were markedly reduced from control values during the critical period of development (days 12 thru 16 of gestation). The activities of the electron transport systems of placental tissue and maternal heart tissue were unaffected during the same period, suggesting that fetal tissue is particularly susceptible to the effects of riboflavin deficie
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