This year the interest in sugar-induced cataract, especially diabetic cataract, continued with increased attention to glycation (the reaction of sugars with lens proteins), and the aldose reductase hypothesis has been further undermined. Oxidation and various experimental models continued to receive attention. Research on human cataracts concentrated on modifications of the proteins and on glutathione, which protects the proteins. Investigations of anticataract drugs, especially of those designed to inhibit aldose reductase and of aspirin-like analgesics, continued. Diabetic cataract in rats was delayed or prevented by aspirin, acetaminophen, ibuprofen, and a new “aldose reductase inhibitor.” Curiously, aldose reductase inhibitors also prevented naphthalene-induced cataract.