Mechanisms of Visual Loss in Severe Proptosis
作者:
Peter Dolman,
Louis Glazer,
Gerald Harris,
Randall Beatty,
Bruce Massaro,
期刊:
Ophthalmic Plastic and Reconstructive Surgery
(OVID Available online 1991)
卷期:
Volume 7,
issue 4
页码: 256-260
ISSN:0740-9303
年代: 1991
出版商: OVID
关键词: Orbital hypertension;Proptosis;Visual loss
数据来源: OVID
摘要:
Vision loss in orbital hypertension secondary to sudden space-occupying lesions is usually attributed to one of three causes: central retinal artery occlusion, direct compressive optic neuropathy, or compression of optic nerve vasculature. Accepted modes of decompressive therapy include lateral canthotomy and cantholysis; drainage of localized orbital air, hematoma, or abscess; and bony wall decompression. Five cases are presented in which orbital hypertension caused severe proptosis with traction on the optic nerve and tenting of the posterior globe. Another mechanism contributing to visual loss is proposed in these cases: ischemie optic neuropathy due to stretching of nutrient vessels. In these cases, rapid posterior decompression should theoretically be favored to reduce orbital pressure and relieve traction on the optic nerve vasculature.
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