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| 11. |
Effect of Methylcholanthrene or Phenobarbital Pretreatment of Rabbits and Rats on the Extinction Coefficient for Cytochrome P-450 |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 43-48
F.E. Greene,
B. Stripp,
J.R. Gillette,
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摘要:
Phenobarbital (PB) or methylcholanthrene (3-MC) pretreatment of rats or rabbits causes induction of cytochrome P-450. In rabbits neither pretreatmentchanges the extinction coefficient for cytochrome P-450-CO complex when this is estimated from the amount of totalheme, cytochrome b5 and absorbancy at 450 nm. In rats PB pretreatment does not change the extinction coefficient, while 3-MC causes an increase in the average extinction coefficient from 85 to 102 mM–1cm–1
ISSN:0031-7012
DOI:10.1159/000136172
出版商:S. Karger AG
年代:1971
数据来源: Karger
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| 12. |
Zur Frage zentraler Wirkungen von 1,1’-Trimethylen-bis(4-formyl-pyridiniumbromid)-dioxim (TMB4) |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 49-55
H.H. Wellhöner,
W.D. Wiezorek,
C. Morgenstern,
M. Maschhour,
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PDF (757KB)
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ISSN:0031-7012
DOI:10.1159/000135054
出版商:S. Karger AG
年代:1961
数据来源: Karger
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| 13. |
Catecholamine-Independent Glycogenolysis in Brain During Carbon Monoxide Poisoning |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 55-63
C.-J. Estler,
F. Heim,
H.P.T Ammon,
V. Zimmermann,
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PDF (885KB)
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摘要:
In carbon monoxide poisoned mice enhanced glycogenolysis and lactate production in the brain are not prevented by pretreatment of the animals with reserpine or an adrenergic β-receptor blocking agent (Kö 592). Since, furthermore, glycogenolysis is not accompanied by changes in the phosphorylase a content or phosphorylase a/phosphorylase (total) ratio, it becomes unlikely that adrenergic mechanisms play a major role in the stimulation of carbohydrate breakdown during carbon monoxide poisoning. It appears more likely that by analogy to the condition in heart glycogenolysis is stimulated by an activation of phosphorylase b due to changes in the concentration of metabolites which are known to inhibit (ATP, glucose-6-phosphate) or activate (AMP, inorganic phosphate) phosphorylase
ISSN:0031-7012
DOI:10.1159/000136174
出版商:S. Karger AG
年代:1971
数据来源: Karger
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| 14. |
The Role of Potassium Ions in Exercise Hyperaemia |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 56-60
Ingemar Kjellmer,
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PDF (571KB)
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ISSN:0031-7012
DOI:10.1159/000135055
出版商:S. Karger AG
年代:1961
数据来源: Karger
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| 15. |
Buchbesprechungen – Book Reviews – Livres Nouveaux |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 61-64
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PDF (489KB)
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ISSN:0031-7012
DOI:10.1159/000135056
出版商:S. Karger AG
年代:1961
数据来源: Karger
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| 16. |
Book Reviews |
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Pharmacology,
Volume 5,
Issue 1,
1971,
Page 64-64
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PDF (119KB)
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ISSN:0031-7012
DOI:10.1159/000136175
出版商:S. Karger AG
年代:1971
数据来源: Karger
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Volume 5 issue 1