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| 1. |
Cardiovascular disease and hyperlipidaemia |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 41-51
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 2. |
Nutrition and therapeutics |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 89-92
David Roberts,
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 3. |
Genetics and molecular biology |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 92-94
Ruth Morgan,
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PDF (278KB)
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 4. |
Lipid metabolism |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 95-99
Ian Young,
Paul Nicholls,
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PDF (514KB)
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 5. |
Cardiovascular disease and hyperlipidaemia |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 99-102
Ngoc-Anh Le,
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PDF (403KB)
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 6. |
Atherosclerosis: cell biology and lipoproteins |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 103-108
John Giddings,
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 7. |
Clinical chemistry and coagulation |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 108-110
Robert Cramb,
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PDF (289KB)
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 8. |
Cardiovascular disease and hyperlipidaemia |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 249-251
Ronald Krauss,
Y Antero Kesäniemi,
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PDF (334KB)
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ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 9. |
Atherosclerosis and arterial influx of lipoproteins |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 252-257
Børge Nordestgaard,
Lars Nielsen,
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摘要:
Accumulation of atherogenic lipoproteins in the arterial intima is a key event in the development of atherosclerosis. The likelihood of increased lipoprotein accumulation in the intima increases with increasing influx: arterial influx of lipoproteins is increased by increased arterial wall permeability, increased lipoprotein concentration in plasma, and by high blood pressure. On entrance into the arterial intima, lipoproteins can either efflux as intact particles, be retained in the intima, or be degraded by intimal cells. Increased degradation as well as increased retention of lipoproteins in the intima have both been associated with increased development of atherosclerosis. Therefore, low rates of efflux of intact particles, as appears to be the case for large particles like VLDL, IDL, and possibly lipoprotein (a) could potentially promote atherosclerosis.
ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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| 10. |
The mouse as a model for human cardiovascular disease and hyperlipidemia |
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Current Opinion in Lipidology,
Volume 5,
Issue 4,
1994,
Page 258-264
Beverly Paigen,
Andrew Plump,
Edward Rubin,
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摘要:
The mouse has been used as an experimental model for atherosclerosis research for only a short time; however, the sophisticated genetics of this species has resulted in a number of innovative approaches that are not possible with other models. The availability of inbred, congenic, recombinant inbred, and mutant strains has resulted in the discovery of a number of genes affecting atherosclerosis susceptibility. More importantly, the newer genetic technologies such as quantitative trait-loci mapping, transgenic mice, and gene-targeted mice are producing important insights into atherosclerosis. This review, focusing on murine models of cardiovascular disease and hyperlipidemia, will be divided into two parts: naturally occurring models and genetically engineered models.
ISSN:0957-9672
出版商:OVID
年代:1994
数据来源: OVID
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