|
1. |
Periodontal Response After Tooth Movement Into Intrabony Defects† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 197-202
Alan Polson,
Jack Caton,
Anne P. Polson,
Sture Nyman,
John Novak,
Brian Reed,
Preview
|
PDF (10438KB)
|
|
摘要:
The present study was undertakensince conflicting evidence exists regarding the effect of such tooth movement on levels of connective tissue attachment. Localized intrabony pockets were produced around isolated incisors in four rhesus monkeys. The root surfaces were planed to the level of the bone at the base of the angular bony defects. An oral hygiene regime was begun and continued for the remainder of the study. The experimental teeth were moved orthodontically into, and through, the original area of the intrabony defect. Two months after cessation of active tooth movement, block specimens were removed for histologic analysis. Control specimens comprised those teeth with induced periodontal defects, but without tooth movement. In specimens not subjected to tooth movement, angular bony defects were present and epithelium lined the root surface to the apical extent of instrumentation. The alveolar bone adjacent to the orthodontically moved teeth no longer had angular defect morphology. On the pressure side, epithelium lined the root surface, was interposed between root surface and bone and terminated at the apical limit of root instrumentation. On the tension side, the crest of the bone was located apical to the level of root planing, and epithelium lined the instrumented portion of the root surface. It was concluded that orthodontic tooth movement into intrabony periodontal defects was without effect upon the levels of connective tissue attachment.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.197
出版商:Wiley
年代:1984
数据来源: WILEY
|
2. |
A Histometric Evaluation of the Effect of Citric Acid Preparation Upon Healing of Coronally Positioned Flaps in Nonhuman Primates† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 203-212
S. G. Woodyard,
A. J. Snyder,
G. Henley,
R. B. O'Neal,
Preview
|
PDF (18523KB)
|
|
摘要:
Facial recession defectswere created on maxillary canine teeth of sixMacaca irusmonkeys and left untreated and exposed to oral fluids for 6 to 12 weeks. Notches were placed in the exposed root surfaces at the level of the free gingival margins. Following root planing with the addition of topical citric acid application on experimental surfaces, pedicle flaps were coronally positioned over the previously exposed roots. After euthanasia, block sections representing postsurgical time periods of 0, 3, 7, 14, 21, 28 and 42 days were secured and tissues were processed for histologic evaluation. All citric acid‐treated surfaces exhibited new connective tissue attachment of pedicle flaps to previously exposed areas by 14 days with transmission electron micrographs confirming beginning cementum deposition. In contrast, controls demonstrated epithelial migration to, or apical to, reference notches. Although the total number of samples available for statistical comparison was small, a two‐tailedttest for correlated samples showed citric acid application did not result in enhanced clinical root coverage, but did result in significantly greater amounts of new connective tissue attachment (P<0.05, df = 3). Pedicle flap healing against teeth with devital pulps was identical to that seen in teeth with vital pulps, while citric acid application to root‐planed surfaces of vital teeth had no observable effect upon pulpal tissues.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.203
出版商:Wiley
年代:1984
数据来源: WILEY
|
3. |
Relative Resistance of Long Junctional Epithelial Adhesions and Connective Tissue Attachments to Plaque‐Induced Inflammation† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 213-223
Robert H. Beaumont,
Timothy J. O'Leary,
Abdel H. Kafrawy,
Preview
|
PDF (17380KB)
|
|
摘要:
This study compared the resistanceto periodontal disease of the long junctional epithelial adhesion and the naturally occurring dentogingival junction. Two groups were used, each containing three young male beagle dogs with all permanent teeth erupted. Periodontitis was induced around maxillary and mandibular premolars in the experimental dogs over a 42‐day period, using subgingival ligatures and a soft diet. Fourteen days after ligature removal, flaps were reflected, granulation tissue was removed and the roots were planed to the alveolar crest. Reference grooves were placed in the root surfaces at the level of the alveolar bone, the flaps were positioned over the alveolar crests, and sutures were placed. A 60‐day period permitted healing with formation of long junctional epithelial adhesions. During this 116‐day period control dogs were maintained in gingival health by daily brushing and by prophylaxis every 14 days. Both groups had a high level of health (GI scores of 0) at the beginning of the 20‐day combined disease phase. Inflammation was induced in both groups by subgingival ligature placement and a plaque‐promoting diet. Right and left sides of both groups represented separate time intervals within the 20‐day period. Block sections were secured at time of killing and the tissues were prepared for light and fluorescent microscopic evaluation.Mean GI scores and mean probing depths increased similarly in both groups. Tagge index scores of gingival inflammation were higher at the longer time periods in the experimental animals. However, they displayed an intact long junctional epithelial adhesion throughout the study, while control animals frequently showed ulceration of the sulcular epithelium. Neither group showed significant changes in location of the apical cells of the attachment epithelium. Crestal osteoblastic activity, confirmed with Procion labeling, predominated in the experimental animals, while osteoclastic activity predominated in the control. Under the conditions of this study, there appeared to be no appreciable difference in resistance to disease between a long junctional epithelial adhesion and a true connective tissue attachment.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.213
出版商:Wiley
年代:1984
数据来源: WILEY
|
4. |
The Acro‐Osteolysis (Hadju‐Cheney) Syndrome: Review of the Literature and Report of a Case |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 224-229
Carl M. Allen,
Lewis Claman,
Randy Feldman,
Preview
|
PDF (9998KB)
|
|
摘要:
Acro‐osteolysis (hadju‐cheney) syndromeis a rare disorder of bone metabolism characterized by progressive destruction of the distal phalangeal bone, curvature of the spine, aplasia of the facial sinuses, and persistence of the cranial sutures. The purpose of this manuscript is to focus on the description of the oro‐facial and dental manifestations of the disorder.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.224
出版商:Wiley
年代:1984
数据来源: WILEY
|
5. |
The Lateral Periodontal Cyst: A Report of 37 Cases |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 230-234
David Andrew Cohen,
Brad W. Neville,
Douglas D. Damm,
Dean K. White,
Preview
|
PDF (6287KB)
|
|
摘要:
The lateral periodontal cystis a developmental cyst of odontogenic origin which is located along the lateral aspect of a tooth root. Thirty‐seven previously unreported lateral periodontal cysts (LPCs) are presented. Clinical, roentgenographic and histologic features of the LPC are reviewed. Further, the importance of distinguishing the developmental LPC from laterally positioned inflammatory cysts of pulpal or periodontal disease origin and laterally occurring odontogenic keratocysts is stressed.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.230
出版商:Wiley
年代:1984
数据来源: WILEY
|
6. |
Plasma‐Cell Gingivitis: Report of a Case† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 235-241
Richard M. Lubow,
Robert L. Cooley,
Kenton S. Hartman,
Raymond K. McDaniel,
Preview
|
PDF (17911KB)
|
|
摘要:
Awell‐documented caseof plasma‐cell gingivitis is presented. When viewed in a total perspective, the clinical examination, history of usage of a popular mint, laboratory data and histologic examination provide support for this diagnosis. This patient did not exhibit any evidence of glossitis or Cheilitis as is often reported in the literature; however, a positive history to psoriasis was noted. The occurrence of plasma‐cell gingivitis in a patient with documented psoriasis provides some interesting speculation regarding the etiologic picture of this condition.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.235
出版商:Wiley
年代:1984
数据来源: WILEY
|
7. |
Caries Levels in Patients With Juvenile Periodontitis† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 242-246
D. H. Fine,
D. Goldberg,
R. Karol,
Preview
|
PDF (1428KB)
|
|
摘要:
The degree and distributionof tooth decay in a group of juvenile periodontitis (JP) patients were compared with tooth decay in an age‐ and sex‐matched periodontally normal control group. The experimental group was comprised of 13 patients of no more than 20 years of age who were designated as localized juvenile periodontitis patients (LJP) according to the definition of Baer, and 10 patients of no more than 25 years of age who were designated as generalized juvenile periodontitis patients (GJP), as described by Manson and Lehner. Twenty‐three age‐ and sex‐matched patients with no periodontal disease served as the control group. Caries was evaluated by standard clinical and radiographic criteria and quantitated by decayed, missing, filled teeth and surfaces (DMFS). Decay was further categorized as active proximal decay and as total proximal decay, which included both active proximal caries and restored proximal surfaces.A Student'sttest revealed no statistical differences between the LJP and GJP groups. Moreover, there was no difference in the number of missing or filled teeth in the JP group as compared to the control group. However, 17 of the 23 JP patients had no proximal decay, whereas all but two of the 23 control patients had some proximal decay. Moreover, in the JP group (LJP and GJP) the mean values for active proximal decay and total proximal decay were both significantly lower than those in the control group (P<0.001). Thus, a low number of proximal carious lesions were found in the presence of juvenile periodontitis. Furthermore, this unusual pattern of proximal dental disease was significantly different from that of the matched control group. The bacteriologic and immunologic significance of these findings deserves further attention.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.242
出版商:Wiley
年代:1984
数据来源: WILEY
|
8. |
The Effects of Topical Steroidal and Systemic Nonsteroidal Anti‐Inflammatory Drugs on Experimental Gingivitis in Man† |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 247-251
Richard I. Vogel,
Stephen A. Copper,
Lawrence G. Schneider,
David Goteiner,
Preview
|
PDF (1427KB)
|
|
摘要:
Retrospective studies in manand prospective studies in animals have indicated that systemically administered anti‐inflammatory drugs may decrease plaque‐induced inflammation and loss of attachment. The purpose of the present double blind study was to determine the effects of a systemically administered nonsteroidal anti‐inflammatory drug and a topically applied steroidal anti‐inflammatory drug on experimentally produced gingivitis. Eighteen dental students were brought to a state of optimal gingival health and then divided into three groups. One group received placebo gel to apply topically and placebo capsules. A second group received placebo gel and capsules containing sulindac, a nonsteroidal anti‐inflammatory drug. The third group received a topical steroidal gel and placebo capsules. All subjects refrained from home care for 22 days in the maxillary right quadrant. Results of the study indicate that the topical steroidal drug significantly inhibited gingival inflammation while the systemically administered nonsteroidal drug had no apparent effect.
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.247
出版商:Wiley
年代:1984
数据来源: WILEY
|
9. |
Letter to the Editors |
|
The Journal of Periodontology,
Volume 55,
Issue 4,
1984,
Page 252-252
Louis P. Gangarosa,
Preview
|
PDF (221KB)
|
|
ISSN:1049-8885
DOI:10.1902/jop.1984.55.4.252
出版商:Wiley
年代:1984
数据来源: WILEY
|
|