摘要:

AbstractTwo experiments were conducted, employing a 2 × 2 factorial design, to assess both 1)a relatively controlled procedure for instigating aggressive inclinations and 2) a way of measuring interpersonal aggression that is much more subtle than most of the laboratory procedures now being used. In both of these experiments half of the subjects were provoked by exposing them to a self‐esteem‐threatening evaluation ostensibly from a peer.After this “treatment,” in experiment 1 all of the subjects had an opportunity to deliver electric shocks to the supposed other student, much as in many of the laboratory experiments in this area, and then rate that student. Shock intensity was not significantly correlated with the unfavorableness of these latter ratings, as if the subjects had reacted in different ways to the shocks they had delivered.In experiment 2, after the instigating treatment the subjects were allowed to withhold rewards from the other person whenever he made correct guesses on a supposed “ESP task.” In this procedure the behavioral hostility (withholding rewards) was positively correlated with the unfavorableness of the subsequent questionnaire ratings, apparently because the subjects were not fully aware of having express

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<237::AID-AB2480120402>3.0.CO;2-U

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractThe scores of three aggression parameters in a standard‐opponent test correlated significantly with individual androgen concentrations measured in tissue as well as in blood samples taken immediately after the test (bites: r = +.62/+.82, threats: r = + 0.75/+.70, latency: r = −.66/−.53). If the test was performed after 28 days of social isolation no correlations were found. There were also no correlations between behavioural measures and corti

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<249::AID-AB2480120403>3.0.CO;2-K

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractGonadectomized male mice from the CF‐1, CFW, and CD‐1 strains were exposed to different androgens (testosterone, dihydrotestosterone, methyltrienolone) or estrogens (estradiol, diethylstilbestrol) and tested for aggressive behavior. Genetic differences in sensitivity to the aggression‐promoting property of the various treatments were found. CF‐1 mice responded to either androgenic or estrogenic stimulation, CFW males were most sensitive to the estrogen treatments, while CD‐1 males responded to androgen but were relatively insensitive to the aggression‐promoting property of estrogens. These data suggest that there may be multiple aggression‐activating systems and that the functional pathway varies with genotype. Additional experiments examined whether receptor binding in the hypothalamic‐preoptic‐septal region was related to the genetic differences in behavioral responsiveness. The binding data suggested that sensitivity to the aggression‐promoting property of estrogens was related to either a higher concentration of binding sites or to higher affinity binding between estrogen and its receptor. A systematic relationship between dihydrotestosterone binding and behavioral responsiveness to and

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<255::AID-AB2480120404>3.0.CO;2-S

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractTreatment with the phenylpiperazine DU 27716 (fluprazine hydrochloride) inhibited the predatory killing of albino mice by northern grasshopper mice (Onychomys leucogaster) and of frogs by rats. This treatment had no effect on cricket predation by grasshopper mice or worm predation by rats. The prey‐specific effect of fluprazine did not seem to result from a decreased tendency to attack nor was its effect restricted to prey showing characteristic rodent defensive responses. Rather, the drug seemed to increase fearfulness when the prey exhibited vigorous defensive behavior. It is possible that increased fearfulness induced by treatment with this drug may also contribute to its antioffensive effect during conspecific encounter

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<267::AID-AB2480120405>3.0.CO;2-I

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractThe effects of serotonin were studied on locust‐killing behavior of mice from low (DD) and high (CBA) predatory aggressive strains. 5‐HTP injected intraperitoneally (50 and 100 mg/kg) or 5‐HT administered into the lateral ventricle (10 μg) significantly reduced locust‐killing behavior in highly aggressive CBA mice. Imipramine (20, 30, and 40 mg/kg) elicited a dose‐dependent inhibitory effect on predatory behavior. Fluoxetine (10 and 20 mg/kg) alone had a slight influence on locust‐killing behavior but potentiated the action of the subthreshold dose of 5‐HTP (25 mg/kg). Pretreatment with the blocker of 5‐HT2type receptors methysergide (2 mg/kg) abolished the inhibitory effect of 5‐HTP. These finding indicate that serotonin of the brain exerts an inhibitory effect on predatory behavior in mice. In contrast, neither lesion of the dorsal raphe nucleus (although significantly depleting the brain serotonin) nor treatment with methysergide (2 mg/kg) induced locust‐killing behavior in weakly aggressive DD mice. Low predatory aggressiveness in DD mice is suggested to be related to the low tonus of the mechanisms activating killing behavior rather than to excessive serotonergic i

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<277::AID-AB2480120406>3.0.CO;2-E

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractIt was predicted from the dopamine‐opiate system hypothesis that aggression induced by light tail pressure would be modified by exposure of the animals to radiofrequency electromagnetic energy. In a series of three experiments, it was found that there was a substantial decrease in aggressive behavior during exposure to EM energy at incident power densities as low as 50 μW/cm2. This is consistent with previously published reports that indicate the dopamine and opiate systems of the brain are influenced by exposure to low‐intensity EM energy. It would appear that EM energy exposure could be used as a probe in the study of aggression and in relating it to brain chemistry. Since this energy has become ubiquitous in the environment, and quite uneven in distribution, there may be implications for understanding aggressive behavior from a clinical standpoint as well as from the standpoint of necessary controls in experim

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<285::AID-AB2480120407>3.0.CO;2-G

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractFemale rats, chronically treated with Testosterone Propionate (TP), were injected with Fluprazine (DU 27716) or saline and tested for social aggression, masculine and feminine sexual behavior. Fluprazine‐treated females were less aggressive than saline‐treated females, as indicated by a shift from offensive to less offensive parameters of aggression. At the same time, mounting in Fluprazine‐treated females was almost totally abolished, both in aggression tests and in tests for sexual behavior. Feminine sexual responses increased during aggressive encounters but were slightly inhibited when females were confronted with sexually active males. Females treated with Fluprazine and tested for mounting with a receptive female showed a substantial increase of offensive aggression directed at the receptive stimulus female. It is concluded that Fluprazine does not selectively inhibit offensive aggression in TP‐treated fema

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<293::AID-AB2480120408>3.0.CO;2-I

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

摘要:

AbstractMale habitually violent and impulsive offenders were studied by means of the glucose tolerance tests (GTTs) and insulin measurements. Both in intermittent explosive disorder and in violent antisocial personality, there was a tendency for there to be reactive hypoglycemia. There was a more rapid decline of glucose from hyperglycemia and more rapid return from hypoglycemia to the original basal values in intermittent explosive disorder than in violent antisocial personality. Consistent with this, the enhanced insulin secretion started rapidly but lasted a short time in intermittent explosive disorder.

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<303::AID-AB2480120409>3.0.CO;2-L

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<311::AID-AB2480120410>3.0.CO;2-F

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY

ISSN:0096-140X    

DOI:10.1002/1098-2337(1986)12:4<::AID-AB2480120401>3.0.CO;2-3

出版商:Wiley Subscription Services, Inc., A Wiley Company    

年代:1986

数据来源: WILEY