ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

摘要:

The Occupational Medicine Forum presents questions of general interest received by the ACOEM Committee on Occupational Medical Practice. Answers usually are prepared by the Committee, representing the composite best judgment of its members. Such replies do not necessarily represent an official position of the American College of Occupational and Environmental Medicine. In some cases, the responses will be prepared at the Committee's request by consultants with special qualifications in specific fields. Questioners are not named. Readers who wish to submit questions to the Committee should address inquiries to: Committee on Occupational Medical Practice, American College of Occupational and Environmental Medicine, 55 W. Seegers Rd, Arlington Heights, IL 60005.

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

摘要:

This article summarizes, compares, and contrasts the definition, sources, magnitude, effect modifiers, and strategies of reduction of the healthy worker effect (HWE), based on the opinion expressed in the papers of nine contributors who responded to the request of the Industrial Disease Standards Panel (IDSP), Ontario, Canada. It provides an insight into the complex issues relating to the HWE. In addition, the catalog of 15 strategies to reduce the HWE is deemed to be useful for investigators in occupational epidemiology.

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID

摘要:

In a retrospective comparison of two worker cohorts with elevated total antibody against trimellitic anhydride (TMA) conjugated to human serum albumin (TM-HSA), IgG subclass antibodies against TMA were studied in 19 workers with and 12 workers without TMA-induced immunologic lung disease. The main outcome measures were ELISA index of IgG 1, IgG2, IgG3, and IgG4 against TM-HSA. There were no statistically significant differences in levels of any IgG subclass between these two groups. Neither were there any statistically significant differences when workers without lung disease were compared with subgroups of workers with lung disease, such as late respiratory systemic syndrome (n= 8), asthma/rhinitis (n= 6), or both (n= 5). In TMA workers with elevated total antibody against TM-HSA, IgG subclasses against TM-HSA in workers with TMA-induced immunologic lung disease were not different from workers without disease.

ISSN:0096-1736    

出版商:OVID    

年代:1992

数据来源: OVID